Just How Toxic Is The Herbicide Used In The Yaquina Basin? Our observations are not intended to be a criticism the property owners who use herbicide to rid their property of unwanted vegetation.
We do not know the unintended consequences of consuming shellfish contaminated with the unknown levels of herbicide!!! We do not recommend consumption of the clams dug from Yaquina Bay, especially eaten by children or the elderly. However the State of Oregon is another matter altogether: they continue to allow the contamination of our watersheds with herbicides and other contaminates.
Toxins and contaminates bio-accumulate in clams - Do not feed contaminated shellfish to your children or the elderly.
I use to dig clams at Sally's Bend in Yaquina Bay. Then the new neighbor sprayed the black berry vines on his property ending my clam digging adventure at Sally's Bend because the clams were contaminated from the herbicide runoff into the tidal flats at Sally's Bend.
The black berry vines at the NW Natural Gas Plant were sprayed with herbicide. The runoff from the spraying had to contaminate the clams in the tidal flats adjacent to the Gas Plant. Glyphosate use is rampant is the watersheds of Oregon's State Waters.
Meanwhile up river a rancher sprayed the black berry vine to get rid of them. Yaquina River use to produce some nice fat crawdads but I failed to catch any this year... This answered my question because I had no intention of eating them because of the high number of creosote rail road ties discarded along the Yaquina River as shown in the photo panel below.
I did a little research on spraying blackberries and they don't recommend doing it during the growing season because they will grow back. They said it should be done late fall or early winter when the leaves are going into fall yellow colors. This spraying over and over at the wrong time of the year is another problem for shellfish and other animals who live within these chemical zones. A little research goes along way.
Unfortunately for me I no longer trust eating bay clams. The State of Oregon Gippo shellfish testing program that rarely test our bays is why I won't eat them anymore.
Considering all the sources of contamination in the Yaquina Basin watershed is there wonder why the salmon runs are in decline.
The following photos of Creosote railroad ties were taken from a series of photographs from over thirty piles of creosote railroad ties discarded along the Yaquina River. We filed a complaint with every level of Oregon's State Government from Governor Brown down.
WE RECOMMEND AGAINST THE CONSUMPTION OF CRAWDADS OR SHELLFISH TAKEN FROM THE YAQUINA RIVER WATERSHED BASIN. CONSIDER, THE TIP OF THE ICEBERG DID NOT SINK THE TITANIC: THE PART OF THE ICEBERG UNDERWATER DID!!!
The State at the direction of Governor Brown by doing nothing refuses to cleanup the Creosote Waste Dump scattered along the railroad the runs along the Yaquina River. Over 30 piles of discarded creosote impregnated R/R ties dumped next to the Yaquina River. View the video clip. We have asked every level of State Government to compel the RR to compel the RR to pickup their creosote soaked RR ties. Is there a compelling reason why Governor Brown allows the continuing pollution of the Yaquina River?
Not covered in this brief piece are the history of creosote nor any of the marine applications.
Creosote is a general term covering coal tar creosote, coal tar, and coal tar pitch. Coal tar creosote is the most common mixture, and is widely used as a wood preservative in the U.S. As many as 10,000 chemicals may comprise this mixture.
• The primary chemicals of concern within creosote compound —with harmful health effects—are polycyclic aromatic hydrocarbons (PAHs), phenols, and creosols. Of these three, PAHs are the most common ingredient.
• PAHs are organic compounds, and in the context of creosote (and in general) they are divided into two categories: low molecular weight PAH compounds (low PAH) and high molecular weight PAH compounds (high PAH).
• Chemicals in creosote break down in water very slowly. They tend to cling to particles of matter, and as such, sediments are considered the primary location for these contaminants to collect in aquatic environments. Many PAHs do not migrate far from the point of contamination, and accumulate at that primary location.
• The rate of absorption of a particular compound is variable and depends on several factors (e.g., exposure, degradation, and oxidation).
• Microscopic breakdown of some of these toxins can occur at the water/sediment interface; high PAHs can resist degradation more than low PAHs. In sediment with less oxygen, degradation of both high and low PAHs decreases, and may persist in the sediments for an indefinite period.
• Intake by living organisms depends on a particular chemical compound, but organisms that live in or are attached to the sediments can uptake many of the PAHs as they ingest sediment or as they eat other organisms with concentrations of the chemicals.
Toxicity of creosote
The toxic effects of organic contaminants (such as PAHs) depends on several factors, including the route of exposure, duration and concentration, chemical composition, organism sensitivity, life stage affected, organism potential for detoxification/excretion, and the physical condition of a particular organism during exposure.
• In general, these chemical compounds vary widely in toxicity. For some organisms, low PAHs are acutely toxic but may be considered non-cancer causing. High PAHs however, are not as toxic, but to many organisms—such as fish, birds, amphibians, mammals—can cause cancer, mutation or malformation of embryo/fetus.
• These toxins quickly accumulate in an organism, but most organisms can also rapidly metabolize and eliminate them. Most fish tissue contains relatively low concentrations of these toxins, and accumulation is usually short term. Two other processes are more Washington State Department of Natural Resources ∙ Aquatic Resources Division ∙ 1 of 2 ∙ Updated - 10/2/2013
common: biodegradation (decay and absorbed by environment) and biotransformation (chemical compound alteration by enzymes).
• PAHs generally are not expected in higher order organisms; organisms such as fish have the potential to metabolize and excrete PAHs.
• These toxins can accumulate in tissues of mollusks and other benthic invertebrates that do not metabolize as efficiently. An increase in concentration can result within organisms with higher fat. Reproduction may be inhibited or death may occur.
• For some fish species, this sediments contamination is linked to adverse impacts such as reproductive impairment, suppressed immune function, liver lesions and fin abnormalities. In addition, embryonic development of the Pacific herring has been shown to be negatively affected by diffusible components of weathered creosote pilings.
• Exposure of fertilized salmon eggs to low levels (1-10 ppb in water; ~1000 ppb in oiled gravel) of total PAHs from weathered oil is linked to reduced adult returns 2 years after exposure--possibly due to impaired cardiac function.
• Juvenile salmon migrating through urban estuaries show reduced disease resistance and increased PAH exposure, and similar results are seen with PAH exposed animals in lab studies.
• Juvenile salmon migrating through urban estuaries show changes in growth and metabolism, and similar results are seen with PAH exposed animals in lab studies. Fish at higher doses experience delayed mortality several months after exposure ended.
Johnson, L.L., M.R. Arkoosh, C.F. Bravo, T.K. Collier, M.M. Krahn, J.P. Meador, M.S. Myers, W.L. Reichert, and J.E. Stein 2007. The Effects of Polycyclic Aromatic Hydrocarbons in Fish from Puget Sound, Washington. The Toxicology of Fishes: Chapter 22.
Vines, C.A., T. Robbins, F.J. Griffin, and G.N. Cherr. 2000. The effects of diffusible creosote-derived compounds on development in Pacific herring (Clupea pallasi). Aquatic Toxicology: 51, pp 225-239.
Agency for Toxic Substances and Disease Registry (ATSDR). 2002. Toxicological profile for creosote. Atlanta, GA: U.S. Department of Health and Human Services, Public Health Service.
EVS Environmental Consultants. 2003. Status, Trends and Effects of Toxic Contaminants in the Puget Sound Environment. Prepared for Puget Sound Action Team, Olympia, Washington. EVS Project No. 02-1090-01.
Video: Chemical Found In Weed Killer Could Increase Your Risk Of Cancer By About 41 Percent: Study (Courtesy: Veuer)
Glyphosate, an herbicide that remains the world's most ubiquitous weed killer, raises the cancer risk of those exposed to it by 41%, a new analysis says.
Researchers from the University of Washington evaluated existing studies into the chemical -- found in weed killers including Monsanto's popular Roundup -- and concluded that it significantly increases the risk of non-Hodgkin lymphoma (NHL), a cancer of the immune system.
"All of the meta-analyses conducted to date, including our own, consistently report the same key finding: exposure to GBHs (glyphosate-based herbicides) are associated with an increased risk of NHL," the authors wrote in a study published in the journal Mutation Research.
The potential carcinogenic properties of glyphosate are the subject of widespread scientific debate. The US Environmental Protection Agency said in a 2017 draft risk assessment that the herbicide "is not likely to be carcinogenic to humans," while the European Food Safety Authority maintains a similar stance. Bayer, which acquired Monsanto in 2018, said the same year that glyphosate is a "safe and efficient weed control tool."