|
|
Post by Admin on Oct 20, 2015 2:26:23 GMT
From: WILLIAM LACKNER
6:58 PM
To: Caren Braby, Rep.DavidGomberg
Dear Caren Braby, Bill Lackner here. Refer to Washington State's news release on June 5th of this year using the link below. My question is, "How did the State of Oregon resolve the closing of crabbing inside the Lower Columbia River Estuary by Washington State?" Did I miss something here?
Thank you, William Lackner for the Clam Diggers Association of Oregon.
wdfw.wa.gov/news/jun0515a/
"The Washington Department of Fish and Wildlife (WDFW) closed crab fishing today (June 5) in coastal waters stretching from the Washington/Oregon border north to Point Chehalis. This area includes Marine Area 1 (Columbia River) and the portion of Marine Area 2 (Westport) south of Point Chehalis, as well as the crabbing area inside the Columbia River and inside Willapa Bay."
William Lackner
|
|
|
|
Post by Admin on Oct 21, 2015 3:11:50 GMT
We thank Caren Brabry for her prompt response to our inquiry. On the face of the information we will assume that Washington State officials are exercising caution when dealing with Domoic Acid. Hopefully we will be on top of this the next time it occurs. Theses are groundbreaking events and crabbers and clam diggers need to be aware of these circumstances of this type of events. Bill
Hello Mr. Lackner,
Thank you for your questions and interest.
To briefly answer your questions, Dungeness crab samples have been collected in the Columbia River by both Washington and Oregon. These samples have not demonstrated a domoic acid (DA) signal to warrant fishery closures (true for both the WA and OR sampling efforts in the river).
Washington did close all of its waters (including its jurisdiction within the Columbia River), due to extremely elevated levels of DA in Dungeness crab collected offshore of WA and in some bays north of the Oregon-Washington border.
Oregon has not closed Oregon waters because we have sampled Dungeness crab in the Columbia River and along the open coast (and in select bays) and have not measured DA levels that warranted fishery closure or even health advisories.
While it may seem a bit odd, Washington has closed their waters to harvest while Oregon’s waters adjacent to them remain open.
We continue to monitor DA levels for Oregon waters and coordinate with both Washington and California on DA results in clams and crabs, given the abnormally high levels of phytoplankton and DA that we have seen this year.
Sincerely, Caren
Caren E. Braby, PhD
Marine Resources Program Manager
Oregon Department of Fish and Wildlife
2040 SE Marine Science Drive
Newport, OR 97365
(541) 867-0300 x 226
(541) 961-5352 (cell)
Caren.e.braby@state.or.us
www.dfw.state.or.us/MRP/
|
|
|
|
Post by Admin on Oct 21, 2015 3:36:09 GMT
The following study suggest that Domoic Acid may be far more harmful to humans than the State and Federal authorities have stated. We have asked the ODA to reconcile the differences between the findings of the study and the threshold level for issuing an alert and they refused.. Bill
Characterization of renal toxicity in mice administered the marine biotoxin domoic Acid.
Funk JA1, Janech MG1, Dillon JC1, Bissler JJ2, Siroky BJ2, Bell PD3.
Author information
Abstract
Domoic acid (DA), an excitatory amino acid produced by diatoms belonging to the genus Pseudo-nitzschia, is a glutamate analog responsible for the neurologic condition referred to as amnesic shellfish poisoning. To date, the renal effects of DA have been underappreciated, although renal filtration is the primary route of systemic elimination and the kidney expresses ionotropic glutamate receptors. To characterize the renal effects of DA, we administered either a neurotoxic dose of DA or doses below the recognized limit of toxicity to adult Sv128/Black Swiss mice. DA preferentially accumulated in the kidney and elicited marked renal vascular and tubular damage consistent with acute tubular necrosis, apoptosis, and renal tubular cell desquamation, with toxic vacuolization and mitochondrial swelling as hallmarks of the cellular damage. Doses≥0.1 mg/kg DA elevated the renal injury biomarkers kidney injury molecule-1 and neutrophil gelatinase-associated lipocalin, and doses≥0.005 mg/kg induced the early response genes c-fos and junb. Coadministration of DA with the broad spectrum excitatory amino acid antagonist kynurenic acid inhibited induction of c-fos, junb, and neutrophil gelatinase-associated lipocalin. These findings suggest that the kidney may be susceptible to excitotoxic agonists, and renal effects should be considered when examining glutamate receptor activation. Additionally, these results indicate that DA is a potent nephrotoxicant, and potential renal toxicity may require consideration when determining safe levels for human exposure.
Copyright © 2014 by the American Society of Nephrology.
|
|
